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by Akademie für Potenzialentfaltung und Persönlichkeitsentwicklung
€62.70 – €100.36

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Persönlichkeitsentwicklung mit Dr. Akuma Saningong

Sat, March 24, 2018, 5:30 PM – 11:30 PM CET

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Sat, March 24, 2018

5:30 PM – 11:30 PM CET


Mindspace Hamburg

Rödingsmarkt 9

20459 Hamburg


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Event description

Du bist der CEO und Pilot Deines Lebens.

Persönliche Entwicklung ist der Glaube daran, dass Du den Aufwand, die Zeit und die Energie wert bist, um Dich selbst zu entwickeln.

Ziel dieses Events ist Dich daran zu erinnern, dass Du ein wertvoller und wundervoller Mensch bist und die Welt ist ein besserer Platz, weil Du existierst. Wir zeigen und erläutern Dir anhand eines inspirierenden Keynote-Talks, dass nahezu alles möglich ist. Wenn Du eine Einstellung vertrittst, die für Alles und Nichts gebunden ist, kannst Du Dein unendliches Potenzial entfalten.

Im weiteren Verlauf des Abends tauchen wir auch in das Thema der Epigenetik ein. Was hat sie mit Deiner Persönlichkeitsentwicklung zu tun? Gleiche Gene bedeuten nicht gleiches Schicksal. Lass Dich überraschen, was dahinter steckt.

Im Zuge dieses einzigartigen Events merkst Du , wie wichtig und mächtig Du bist. Des Weiteren lernst Du zu verstehen, dass Deine Gedanken Deine Realität kreieren.

Ohne persönliches Wachstum, kannst Du nie Dein volles Potenzial entfalten können. Du musst Dich selbst verstehen und kennenlernen, um zu wachsen. Wenn Du die Welt verbessern willst, dann verbessere zuerst Dich selbst.

Du hast es in der Hand. Mach was draus!


17:30 : Einlass + Akkreditierung sowie Snacks

18:30: Einführung Bano Diop

18:30 :

18:40: Persönlichkeitsentwicklung mittels Quantum Denken – Dr. Akuma Saningong

19:40: Pause + Snacks + Getränke


20:20: Epigenetik – Der Code zu Deiner Persönlichkeitsentwicklung – Dr. Akuma Saningong

21:20: Pause 10 min


22:00: Eröffnung Networking + Party

-open End

Das größte Geschenk, welches Du anderen machen kannst, ist daher, Dich selbst weiterzuentwickeln. Und wenn Du etwas ändern willst, musst Du bei Dir selbst anfangen.

Sichere Dir jetzt Dein Ticket für einen Moment der Potenzialentfaltung und puren Inspiration! Unter Gleichgesinnten behältst Du diesen Abend lange in Erinnerung.

PS: Das Event ist ein Feuerwerk an Networking und die Verbreitung von guter Energie unter Gleichgesinnten. Wir freuen uns schon auf Dich.
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Jeremy D. Schmahmann
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Stroke. 2003; 34: 2264-2278
Jeremy D. Schmahmann


This article reviews the anatomy, connections, and functions of the thalamic nuclei, their vascular supply, and the clinical syndromes that result from thalamic infarction.

Thalamic nuclei are composed of 5 major functional classes: reticular and intralaminar nuclei that subserve arousal and nociception; sensory nuclei in all major domains; effector nuclei concerned with motor function and aspects of language; associative nuclei that participate in high-level cognitive functions; and limbic nuclei concerned with mood and motivation. Vascular lesions destroy these nuclei in different combinations and produce sensorimotor and behavioral syndromes depending on which nuclei are involved. Tuberothalamic territory strokes produce impairments of arousal and orientation, learning and memory, personality, and executive function; superimposition of temporally unrelated information; and emotional facial paresis. Paramedian infarcts cause decreased arousal, particularly if the lesion is bilateral, and impaired learning and memory. Autobiographical memory impairment and executive failure result from lesions in either of these vascular territories. Language deficits result from left paramedian lesions and from left tuberothalamic lesions that include the ventrolateral nucleus. Right thalamic lesions in both these vascular territories produce visual-spatial deficits, including hemispatial neglect. Inferolateral territory strokes produce contralateral hemisensory loss, hemiparesis and hemiataxia, and pain syndromes that are more common after right thalamic lesions. Posterior choroidal lesions result in visual field deficits, variable sensory loss, weakness, dystonia, tremors, and occasionally amnesia and language impairment.

These vascular syndromes reflect the reciprocal cerebral cortical-thalamic connections that have been interrupted and provide insights into the functional properties of the thalamus.

In the first detailed account of the behavioral consequences of thalamic hemorrhage, Fisher 1 described 13 patients of a total of 102 cases of intracerebral hemorrhage pathologically studied. Sensory deficits were associated with neglect (“modified anosognosia and hemiasomatognosia”), “dysphasia” was global and moderate in severity, and confusion, delirium, visual hallucinations, and peduncular hallucinosis taking the form of colorful objects, animals, and flowers were described. Later reports of thalamic hemorrhage confirmed and extended the relationship between thalamic lesions and cognitive deficits. 2–5 Hemorrhage is seldom confined to the thalamus itself, however, and remote effects include pressure of the hemorrhagic mass, effect of surrounding edema on adjacent structures, and toxic effects of the blood products. Similarly, accounts of deficits in adults 6 and children 7 with thalamic tumors have helped to shape the understanding of the behavioral manifestations of thalamic lesions, but the structure-function analysis is too complex to establish with certainty that the behavior is a consequence of involvement of the thalamus alone. A patient with a penetrating injury of the left thalamus (intralaminar, dorsomedial, ventral lateral, and ventral anterior nuclei and mamillothalamic tract) developed amnesia for verbal material, but the lesion also involved the posterior hypothalamus and both mamillary bodies. Donald J Pliner Suede Wedge Sandals Cheap Sale Sneakernews 9kXdREd
Conclusions concerning thalamic functions in humans have also been derived from thalamotomy and thalamic stimulation studies (referred to below) and more recently from functional imaging studies that fall outside the scope of this review.

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